N-terminal tyrosine phosphorylation of Cav-2 is certainly activated by inhibition of tyrosine phosphatases in mouse and individual ECs and suppressed by pharmacological inhibition of Src and Abl kinases in individual ECs Due to newly described here functional need for tyrosine phosphorylation in regulating TGF- signaling and function in ECs, next we’ve examined if tyrosine 19 and/or 27 phosphorylation of Cav-2 is regulated in ECs

After 5 weeks, no significant difference between groups was observed in motor function, ADL, or quality of life